It is not normal to fall asleep too often or sleep too often. This can be a sign of a disorder of the nervous system that controls sleep activity. This condition is often referred to as narcolepsy.

Narcolepsy is a neurological disorder that affects a person's sleep control and awareness of what is happening. People with narcolepsy experience sudden and uncontrollable excessive morning and daytime sleepiness. People with narcolepsy may suddenly fall asleep. These sudden sleep attacks can occur during any activity during the day. Not only does it interfere with daily life, it can also harm the patient. You can find easily stimulant, such as Waklert, and Artvigil in your favorite online drugstores, to improve your daily life.

People with narcolepsy may also experience atonic seizures and weakness due to weakness in their muscles.  You can consume Artvigil, such as Artvigil 150 for those conditions.

Symptoms of narcolepsy usually appear by the age of 1025. Narcolepsy can be associated with sleep paralysis, hallucinations, and other symptoms, including weakness or atonic seizures that are weakness or loss of control of the muscles of the face, neck, and knees. Narcolepsy with atonic seizures is called type 1 narcolepsy, and narcolepsy without atonic seizures is called type 2 narcolepsy.  You may consider to buy Waklert, such as Waklert 150 to improve your conditions.

The exact cause of narcolepsy is unknown. However, most people with narcolepsy have low levels of hypocrite. Hippocratic is a chemical in the brain that controls sleep. The cause of narcolepsy with atonic seizures is the lack of chemical hypocrite in the brain. Hippocratic is an important chemical in the regulation of arousal and rapid eye movement (REM) stages. Narcolepsy without atonic seizures has similar symptoms, but the cause is unknown.

What is the cause of narcolepsy?

The exact cause of narcolepsy is unknown. However, experts estimate that some health conditions are associated with the development of this disease. Most people with narcolepsy have low levels of hypocrite. The hypothalamus is a neuron in the hypothalamus that is part of the brain that regulates sleep, appetite, and body temperature.

Hippocratic plays an important role in regulating human sleep rhythms. In addition, hypocrite also functions as a neurotransmitter. Neurotransmitters are compounds that carry signals from one nerve cell to another. Experts believe that reduced hypocrite may affect the appearance of this disorder. Hypothalamic neurons in the hypothalamus usually help the brain keep awake. For unknown reasons, studies have shown that in people with narcolepsy, neurons containing hypocrite lose function and eventually die. Loss of hypocretinic neurons results in decreased alertness and increased daytime sleepiness.

Hypocrite is a neuropeptide used to transmit information between brain cells. The neurotransmitters hypocrite receptor type 1 and hypocrite receptor type 2 play an important role in the regulation of the sleep cycle. Increased levels of this neurotransmitter can lead to increased appetite. Produced in an area of ​​the brain called the hypothalamus, hypocrite is one of the chemical messengers, or neurotransmitters, that affect the brainstem and stimulate food intake, awareness, and energy use. Scientists discovered hypocrites in 1998, and have been researching their role in sleep and arousal, as well as treatments for sleep disorders.

Low or no levels of this hormone are found in people with narcolepsy, a condition that causes excessive daytime sleepiness. The hypocrite peptide is also known as origin. These two terms can be used interchangeably in medical nomenclature. The term hypocrite is generally used when referring to the sleep cycle, and origin is more likely to be used when referring to appetite.

Origin, also called hypocrite, is part of a class of chemicals called stimulating neuropeptide hormones. There are two different peptides with similar chemical structures, one is known as orexin-A (or hypocretin-1) and the other is orexin-B (or hypocretin-2). Orexin deficiency can impair the body's ability to maintain wakefulness and sleep, a difficulty that can lead to narcolepsy.

Fewer than 20,000 cells in the brain are capable of producing this sleep-regulating hormone. The hypothalamus releases peptide molecules of this neurotransmitter into the fluid surrounding the brain and spinal cord. These molecules do not spread to surrounding tissues or the bloodstream. Blood tests can reveal if the levels of this hormone are within normal limits.

Testing for hypocretin-1 levels was performed via a lumbar spinal tap. A long needle is inserted into the spinal cord between the third and fourth vertebrae, and a small amount of cerebrospinal fluid (CSF) is aspirated into the syringe. This fluid is sent to a laboratory to analyze the concentration of neurotransmitters. Test results may indicate a need for a sleep study to confirm a diagnosis of narcolepsy.

Some people with narcolepsy experience a decrease in hypocretin levels by 80-90 percent. According to the National Organization of Rare Disorders, several things can cause a decrease in hypocretin levels in the brain. Decreased hypocretin can be related to the presence of genetic mutations in the body. Some patients with this disease experience changes in the T cell receptor gene. T cells play a role in the human immune system.

In addition, narcolepsy is also associated with a history of heredity in the family. Scientists have found several genes involved in the inheritance of this disease to the next generation. Genetics is thought to be one of the causes of narcolepsy. A person is born with certain genes that put them at a higher risk of developing narcolepsy. In childhood or adolescence, infection can trigger narcolepsy, because the body's immune system attacks special cells in the brain that produce hypocretin. This means, reduced production of hypocretin may be the result of an autoimmune reaction. Thus, narcolepsy is considered an autoimmune disease.

The loss of hypocretin in the brain makes it difficult for a person to stay awake. It also allows the REM stage to occur at inappropriate times. Narcoleptic patients can enter the REM stage directly from the waking state. In well-regulated sleep people, orexin is released when they wake up and helps increase activity in neurons that promote wakefulness and suppress REM sleep – deep sleep when dreams occur.

When a person suffers from narcolepsy, which is characterized by excessive daytime sleepiness and falling asleep without warning, it is because the orex-producing neurons have died. Without orexin, a person with narcolepsy cannot control REM sleep. In some cases, dreams that normally occur during REM sleep can occur when a person wakes up, resulting in dream-like hallucinations.

Researchers believe that narcolepsy is caused in part by an autoimmune attack of orexin neurons. Typically, people with narcolepsy will have elevated levels of antibodies against streptococcus, which is the bacteria that causes strep throat. This suggests that the autoimmune attacks are triggered by inflammation or other winter infections, especially since narcolepsy usually strikes in late spring or early summer.

If hypocretin deficiency causes insomnia, it makes sense that hypocretin can also help keep you awake, right? A drugs named orexin receptor antagonists has developed to treat sleep disorders such as insomnia. This sleep aid targets orexin to function by blocking hypocretin signaling in the brain. Hippocretin serves to awaken and alert people, thereby preventing them from falling asleep.

Orexin receptor agonists differ from other prescription and over-the-counter hypnotics in that they target different, more localized chemical systems in the brain. Suvorexant, sold under the name Belsomra, is the first class of drug available.